Lesson 2: The Root Cause — How ANS Dysfunction Drives Every Symptom
Understand how a single dysfunction can cause fatigue, pain, and other symptoms across ME/CFS, Fibromyalgia, POTS, MCS and related illness.
ANS REWIRE Introductory Module - 2 of 4 complete 
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This Lesson:
+ How the root dysfunction causes all the symptoms of ME/CFS/Fibromyalgia/POTS/MCS & related conditions;
+ How and why this driving dysfunction establishes itself.
After years of feeling dismissed and blaming myself for this illness, your explanation finally made everything click.
For the first time, I understand what’s really happening in my body — and it’s not my fault. It feels incredible to finally have clarity and validation.”
Lesson 2 Viewer
Next Lesson:
+ How the 6 trigger groups perpetuate the illness;
+ The physical triggers that perpetuate the illness;
+ The scientific breakthrough paving the way for recovery.
Wonderful video! Wonderful explanation! It not only gave me understanding but, at last, HOPE!
The six triggers for dysfunction are EXTREMELY eye-opening.
I understand now why you always clarify that ANS Rewire is NOT simply brain retraining.
So many approaches miss this… I feel like this is the last piece of the puzzle!
Looking forward to tomorrow’s lesson and starting the program soon.”
Lesson 3 Viewer
Program Recovery Testimonial:
Although as a Nutritionist I ‘knew’ how to look beyond the symptoms to the root cause of illness, I wasn’t winning my case of CFS.
I was lucky to have found the CFS Recovery site — this gave me hope and direction.
About the same time, I was sent a link by Dan to the ANS REWIRE programme. Because the programme took a scientific and evidence-based approach, I signed up.
It was not that long before I started to record progress, seeing myself reach 71, 72, 73+% and more.
Now 6 months after starting the programme, I’m so much better.
What Dan’s achieved through the ANS REWIRE programme is remarkable and I’ll always be very grateful to him for it.”
Dear Dan,
thank you for all the work. I strongly relate to the explanation of ANS being the root cause for all the crazy symptoms. It makes sense. But what if there's a deeper root cause for the ANS dsfunction? What if it is an autoimmune disease? I ask this because I heard that they (Dr. Scheibenbogen, Berlin) found antibodies against synapses of the ANS in ME/CFS patients.
So, my concern is, could your program help, if there is an autoimmune response involved? How much sense does it make to educate and train the software, if the hardware is constantly sabotaged in the brain?
unfortunately I cannot find out what is going on in my own brain on the cellular level, because I don't have access to the scarce research institutes. But if I knew I have such antibodies I would probably try to get rid of them first. But at this point Nobody knows if every patient has them.
What do you think about the findings?(How) do they still match your explanation?
thank you so much
Eva
Hi Eva,
Glad the resources are helpful for you.
So you raise a valid question, but is there a neat answer?
Unquestionably, there are many many things going on that we don’t fully understand and I would suggest to you that the way the body works is much more complicated than some people suggest (specifically those who suggest they know fully what’s going on 😉)
So autoimmunity issues are not new or uncommon. We see many people have these issues and resolve them as they recover. It is my perception that since the pandemic, autoimmunity is more of problem for the community in general – this is for various reasons.
However, the question in regards to ME/CFS are:
1.) Is this a problem for all people with ME/CFS
2.) Is this a limiting factor for recovery (I am not aware of such evidence)
3.) Are there any effective treatments
The notion that we can fix something with a treatment is from lack of recognition of the complexity of the body. Just like you suggest that there is a hardware to software connection, there is a software to hardware connection, hence the field of Psychoneuroimmunology.
Sorry about the waffly circular answer, but that is the nature of how things work.
In summary, my response is that these kind of findings are not new. It is expected that for a real and complex illness like this that there are many many dysfunctions and hence observable abnormalities. Safe and effective physical interventions and treatments can potentially play an important role in recovery. In the end, we need to focus on doing whatever we can to recover and stay away from going down rabbitholes.
Hope that helps you.
Hi Dan,
I’ve been diagnosed with POTS. Practitioners have said that both my parsympathetic, and my sympathetic systems are both over active. Main and most difficult symptoms that I’ve had for 3 years are tremors, heart bounding, dizziness. Symptoms are worsened when I’m busier, there’s extra noise, after I drive, while attending church service. Do you think that this program will help someone with both psns, and the sns in overdrive?
Hi Mary
Sorry for the late reply – I overlooked this one!
It’s my view that indeed the issue is BOTH ANS and PNS over-reaction in people experiencing ME/CFS/Fibro/POTS.
So yes, I would be fine with you enrolling.
Hi Dan,
I really like your hypothesis on how M.E/CFS manifests & it does encapsulate how many of the dysfunctions arrive. I was initially diagnosed with M.E/CFS, but eventually found out it was POTS that was the driving force of many of the symptoms. Since POTS is directly related to autonomic dysfunction, would you be also gearing your ANS Rewire towards these sets of patients? Thank you for all our your work, cause I know myself how much literature is out there in the field & often contradictory.
Hi Sharon,
Apologies, it has been my error not to be more specific about POTS.
POTS is ME/CFS, it’s just that one symptom is pronounced, hence yet another name.
So yes, ANS REWIRE is specifically designed for POTS/ME/CFS/SEID/Fibromyalgia/MCS/CFIDS/PVFS/EMHS. 🙂
No wonder we have difficulty getting help with this illness, so many names just add to the confusion!
Thanks Dan. There is definitely way too many names for one central cause because there also seems to be many subsets of dysautonomia.
Indeed, there are differences in how the ilness expresses itself, which also varies over time. However, It is still my strong view that a common framework applies. 🙂